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Mouse Models of Cerebral Arteriovenous Malformation.Nielsen, Corinne M; Huang, Lawrence; Murphy, Patrick A; Lawton, Michael T; Wang, Rong A Stroke; a journal of cerebral circulation 2016 Jan;47(1):293-300 Tumor angiogenesis in the absence of fibronectin or its cognate integrin receptors.Murphy, Patrick A; Begum, Shahinoor; Hynes, Richard O PloS one 2015 Jan;10(3):e0120872 Constitutively active Notch4 receptor elicits brain arteriovenous malformations through enlargement of capillary-like vessels.Murphy, Patrick A; Kim, Tyson N; Huang, Lawrence; Nielsen, Corinne M; Lawton, Michael T; Adams, Ralf H; Schaffer, Chris B; Wang, Rong A Proceedings of the National Academy of Sciences of the United States of America 2014 Dec;111(50):18007-12 Alternative splicing of endothelial fibronectin is induced by disturbed hemodynamics and protects against hemorrhage of the vessel wall.Murphy, Patrick A; Hynes, Richard O Arteriosclerosis, thrombosis, and vascular biology 2014 Sep;34(9):2042-50 Notch4 normalization reduces blood vessel size in arteriovenous malformations.Murphy, Patrick A; Kim, Tyson N; Lu, Gloria; Bollen, Andrew W; Schaffer, Chris B; Wang, Rong A Science translational medicine 2012 Jan;4(117):117ra8 Endothelial Notch signaling is upregulated in human brain arteriovenous malformations and a mouse model of the disease.Murphy, Patrick A; Lu, Gloria; Shiah, Steven; Bollen, Andrew W; Wang, Rong A Laboratory investigation; a journal of technical methods and pathology 2009 Sep;89(9):971-82 Endothelial Notch4 signaling induces hallmarks of brain arteriovenous malformations in mice.Murphy, Patrick A; Lam, Michael T Y; Wu, Xiaoqing; Kim, Tyson N; Vartanian, Shant M; Bollen, Andrew W; Carlson, Timothy R; Wang, Rong A Proceedings of the National Academy of Sciences of the United States of America 2008 Aug;105(31):10901-6