Reist, Noreen - Colorado State University - Department of Biomedical Sciences

个人简介

PhD, Stanford University BA, University of California, Berkeley

近期论文

查看导师新发文章（温馨提示：请注意重名现象，建议点开原文通过作者单位确认）

Striegel AR, Biela LM, Evans CS, Wang Z, Delehoy J, Sutton RB, Chapman ER, Reist NE. 2012. Calcium binding by synaptotagmin's C2A domain is an essential element of the electrostatic switch that triggers synchronous synaptic transmission. J Neurosci 32:1253-1260. Paddock BE, Wang Z, Biela LM, Chen K, Getzy MD, Striegel A, Richmond JE, Chapman ER, Featherstone DE, Reist NE, 2011. Membrane penetration by synaptotagmin is required for coupling calcium binding to vesicle fusion in vivo. J Neurosci 31:2248-2257 PMID: 21307261. Loewen CA, Reist NE. 2008. Synaptotagmin: Transducing Ca2+-binding to vesicle fusion. In: Molecular Mechanisms of Neurotransmitter Release (Wang ZW, ed), pp 107-134. Totowa: Humana Press. Paddock BE, Striegel A, Hui E, Chapman ER, Reist NE. 2008. Ca2+-dependent, phospholipid-binding residues of synaptotagmin are critical for excitation-secretion coupling. J Neurosci 28:7458-7466. Tamura T, Hou J, Reist NE, Kidokoro Y. 2007. Nerve-evoked synchronous release and high K+-induced quantal events are regulated separately by Synaptotagmin I at Drosophila neuromuscular junctions. J Neurophysiol 97:540-549. Loewen CA, Lee SM, Shin YK, Reist NE. 2006. Synaptotagmin's C2B polylysine motif facilitates a Ca[2+]-independent stage of synaptic vesicle priming in vivo. Molec Biol Cell 17:5211-5226. Loewen CA, Royer SM, Reist NE. 2006. Drosophila synaptotagmin I null mutants show severe alterations in vesicle populations, but calcium-binding motif mutants do not. J Comp Neurol 496:1-12. Mackler JM, Drummond JA, Loewen CA, Robinson IM, Reist NE. 2002. The C2B Ca[2+]-binding motif of synaptotagmin is required for synaptic transmission in vivo. Nature 418:340-344.