王蔚东 - 中山大学 - 中山医学院

个人简介

1999年初以访问学者身份赴丹麦奥胡斯大学，从事肾脏病理生理学研究，2001年至2004年在奥胡斯大学水盐研究中心攻读医学博士学位，并于2004年获得肾脏病理生理学博士学位并晋升为助理教授。2004年9月以博士后身份赴美国科罗拉多州大学医学院高血压与肾脏疾病科继续从事肾脏的生理和病理生理学研究，先后晋职为研究型讲师和助理教授。2012年2月被中山大学中山医学院高血压研究所聘为教授，博士生导师，全职回国工作。

研究领域

从事肾脏的生理和病理生理学研究

近期论文

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Mai X, Shang J, Liang S, Yu B, Yuan J, Lin Y, Zhang F, Liu Y, Lv X, Li C, Wang W*, Zhou J*. Blockade of Orai1 Store-Operated Calcium Entry Protects Against Renal Fibrosis. J Am Soc Nephrol, 2016 accepted co-corresponding author Wang W, Li C, Yang T, Protection of Nitro-Fatty Acid against Kidney Diseases. Am J Physiol Renal Physiol. 2016 accepted, invited review Li C, Lin Y, Luo R, Chen S, Wang F, Zheng P, Levi M, Yang T, Wang W*. Intrarenal Renin-Angiotensin System Mediates Fatty Acid-Induced ER Stress in the Kidney. Am J Physiol Renal Physiol. 2016, accepted. Wang W, Luo R, Lin Y, Wang F, Zheng P, Levi M, Yang T, Li C. Aliskiren Restores Renal AQP2 Expression during Unilateral Ureteral Obstruction by Inhibiting the Inflammasome. Am J Physiol Renal Physiol, 2015, accepted Li C, Wang W*. Urea transport mediated by aquaporin water channel proteins. Subcell Biochem. 2014;73:227-65. Wang W and Yang T, PPAR-γ Signaling in Podocyte Injury. In: Podocytopathy; Editors, Liu Z-H and He JC, Contrib Nephrol. Basel, Karger, 2014, vol 183, pp 101–111 Wang W*, Qiu L, Howard A, Solis N, Li C, Wang X, Kopp JB, Levi M. Protective effects of aliskiren and valsartan in mice with diabetic nephropathy. J Renin Angiotensin Aldosterone Syst. 2014 Dec;15(4):384-95. Li C, Wang W, Rivard C, Lanaspa M, Summer S, Schrier RW. Molecular mechanisms of angiotensin II stimulation on aquaporin-2 expression and trafficking. Am J Physiol Renal Physiol 2011 May;300(5):F1255-61 Wang W, Li C, Summer SN, Falk S, Schrier RW. Interaction between vasopressin and angiotensin in vivo and in vitro: effect on aquaporins and urine concentration. Am J Physiol Renal Physiol. 2010 Sep; 299(3):F577-84 Li C, Wang W, Summer SN, Falk S, Schrier RW. Downregulation of UT-A1/UT-A3 is associated with urinary concentrating defect in glucocorticoid-excess state. J Am Soc Nephrol. 2008 Oct;19(10):1975-81 Wang W, Li C, Summer SN, Falk S, Wang W, Schrier RW. Role of AQP1 in Endotoxemia Induced Acute Kidney Injury. Am J Physiol Renal Physiol. 2008 Jun;294(6):F1473-80 Li C, Wang W, Summer SN, Westfall TD, Brooks DP, Falk S, Schrier RW. Molecular Mechanisms of Antidiuretic Effect of Oxytocin. J Am Soc Nephrol. 2008 Feb;19(2):225-32; Wang W, Li C, Summer SN, Falk S, Schrier RW. Polyuria of thyrotoxicosis: downregulation of aquaporin water channels and increased solute excretion. Kidney Int. 2007 Nov;72(9):1088-94. Li C, Wang W, Norregaard R, Knepper MA, Nielsen S, Frøkiaer J.Altered expression of epithelial sodium channel in rats with bilateral or unilateral ureteral obstruction. Am J Physiol Renal Physiol. 2007 Jul;293(1):F333-41 Wang W, Praetorius J, Li C, Praetorius HA, Kwon TH, Frøkiær J, Nielsen S. Vacuolar H+-ATPase expression is increased in acid-secreting intercalated cells in kidneys of rats with hypercalcemia-induced alkalosis. Acta Physiol (Oxf). 2007 Apr;189(4):359-68. Li C, Wang W, Summer SN, Cadnapaphornchai MA, Falk S, Umenishi F, Schrier RW. Hyperosmolality in vivo upregulates aquaporin-2 water channel and Na-K-2Cl co-transporter in Brattleboro rats. J Am Soc Nephrol. 2006 Jun;17(6):1657-64. Wang W, Li C, Summer SN, Falk S, Cadnapaphornchai MA, Chen YC, Schrier RW. Molecular analysis of impaired urinary diluting capacity in glucocorticoid deficiency. Am J Physiol Renal Physiol. 2006 May;290(5):F1135-42. Wang W, Li C, Nejsum LN, Li H, Kim SW, Kwon TH, Jonassen TE, Knepper MA, Thomsen K, Frokiaer J, Nielsen S. Biphasic effects of ANP infusion in conscious, euvolumic rats: roles of AQP2 and ENaC trafficking. Am J Physiol Renal Physiol. 2006 Feb;290(2):F530-41. Wang W, Li C, Kwon T-H, Miller RT, Knepper MA, Frokiaer J, Nielsen S. Reduced expression of renal Na+ transporters in rats with PTH-induced hypercalcemia. Am J Physiol Renal Physiol. 2004, Mar;286(3):F534-45 Wang W, Li C, Kwon TH, Knepper MA, Frokiaer J, Nielsen S. AQP3, p-AQP2, and AQP2 expression is reduced in polyuric rats with hypercalcemia: prevention by camp-PDE inhibitors. Am J Physiol Renal Physiol. 2002 Dec;283(6):F1313-25