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Macrophage-like U937 cells recognize collagen fibrils with strain-induced discrete plasticity damage. Veres SP, Brennan-Pierce EP, Lee JM. J. Biomed. Mater. Res. A. (2014). Mechanically overloading collagen fibrils uncoils collagen molecules, placing them in a stable, denatured state. Veres SP, Harrison JM, Lee JM. Matrix Biol.(2014) 33:54-9. Repeated subrupture overload causes progression of nanoscaled discrete plasticity damage in tendon collagen fibrils. Veres SP, Harrison JM, Lee JM. J. Orthop. Res. (2013) 31(5):731-7. Cross-link stabilization does not affect the response of collagen molecules, fibrils, or tendons to tensile overload. Veres SP, Harrison JM, Lee JM. J. Orthop. Res. (2013) 31(12):1907-13. Designed to fail: a novel mode of collagen fibril disruption and its relevance to tissue toughness. Veres SP, Lee JM. Biophys. J. (2012) 20;102(12):2876-84. Differences in collagen cross-linking between the four valves of the bovine heart: a possible role in adaptation to mechanical fatigue. Aldous IG, Veres SP, Jahangir A, Lee JM. Am. J. Physiol. Heart Circ. Physiol. (2009) 296(6):H1898-906.