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Chronic exposure to neonicotinoids increases neuronal vulnerability to mitochondrial dysfunction in the bumblebee (Bombus terrestris). Moffat C, Pacheco J G, Sharp S, Samson A J, Bollan K A, Huang J, Buckland S T, Connolly C N. 2015. FASEB. 5-HT cellular sequestration during chronic exposure delays 5-HT3 receptor resensitization due to its subsequent release. Hothersall J D, Alexander A, Samson A J, Moffat C, Bollan K A, Connolly C N. 2014. Journal of Biological Chemistry. Prolonged inhibition of 5-HT3 receptors by palonosetron results from irreversible binding rather than inducing receptor internalisation. Hothersall J. D*, Moffat C*, Connolly C N. 2013. British Journal of Pharmacology * Joint first authorship. Exposure to cholinergic pesticides causes mushroom body neuronal inactivation and deficits in olfactory learning in honeybees. 2013. Palmer M J, Moffat C, Saranzewa N, Harvey J, Wright G A, Connolly C N. Nature Communications. Exposure to acetylcholinesterase inhibitors alters the physiology and motor function of honeybees. Williamson S M, Moffat C, Gommersall M A E, Saranzewa N, Connolly C N, Wright G A. 2013. Frontiers in Invertebrate Physiology The microsporidian parasites Nosema ceranae and Nosema apis are widespread in honeybee (Apis mellifera) colonies across Scotland. 2012. Bollan K.A, Hothersall J.D, Moffat C, Durkacz J, Saranzewa N, Wright G.A, Raine N.E, Highet F, Connolly C N. Parisitology Research.