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HO J, PELZEL C, BEGITT A, MEE M, ELSHEIKHA HM, SCOTT DJ and VINKEMEIER U, 2016. STAT2 Is a Pervasive Cytokine Regulator due to Its Inhibition of STAT1 in Multiple Signaling Pathways. PLoS Biology. 14(10), e2000117 NYIRENDA MH, MORANDI E, VINKEMEIER U, CONSTANTIN-TEODOSIU D, DRINKWATER S, MEE M, KING L, PODDA G, ZHANG G, GHAEMMAGHAMI A, CONSTANTINESCU CS, BAR-OR A and GRAN B, 2015. TLR2 Stimulation Regulates the Balance between Regulatory T Cell and Th17 Function: A Novel Mechanism of Reduced Regulatory T Cell Function in Multiple Sclerosis. Journal of Immunology. 194(12), 5761-5774 PELZEL, CHRISTIN, SCOTT, DAVID JAN, HO, JOHNATHAN ON KAY and VINKEMEIER, UWE, 2015. Quantitative characterisation of STAT2 dimerisation CYTOKINE. 76(1), 83-83 ANDREAS BEGITT, MATHIAS DROESCHER, THOMAS MEYER, CHRISTOPH D. SCHMID, MICHELLE BAKER, FILIPA ANTUNES, KLAUS-PETER KNOBELOCH, MARKUS R. OWEN, RONALD NAUMANN, THOMAS DECKER and UWE VINKEMEIER, 2014. STAT1-cooperative DNA binding distinguishes type-1 and type-2 interferon signaling Nature Immunology. 15, 168-176 BONHAM AJ, WENTA N, OSSLUND LM, PRUSSIN AJ, VINKEMEIER U and REICH NO, 2013. STAT1:DNA sequence-dependent binding modulation by phosphorylation, protein:protein interactions and small-molecule inhibition. Nucleic Acids Research. 41(2), 754-63 WENTA N and VINKEMEIER U, 2013. Characterization of STAT self-association by analytical ultracentrifugation. Methods in Molecular Biology (Clifton, N.J.). 967, 203-24 PELZEL, C., BEGITT, A., WENTA, N. and VINKEMEIER, U., 2013. Evidence against a role for β--Arrestin1 in STAT1 dephosphorylation and the inhibition of interferon-γ signaling Molecular Cell. 50(1), 149-156 SPEIL J, BAUMGART E, SIEBRASSE J, VEITH R, VINKEMEIER U and KUBITSCHECK U, 2011. Activated Stat1 Transcription Factors conduct distinct saltatory Movements in the Cell Nucleus. Biophysical Journal. 101(11), 2592-600 DROESCHER M, BEGITT A and VINKEMEIER U, 2011. Paracrystals of STAT Proteins and their Dissolution by Sumo: How reduced Transcription Factor Solubility increases Cytokine Signaling. Oncotarget. 2(7), 527-8 BEGITT, A., DROESCHER, M., KNOBELOCH, K. and VINKEMEIER, U., 2011. SUMO conjugation of STAT1 protects cells from hyperresponsiveness to IFNγ Blood. 118(4), 1002-1007 ANTUNES F, MARG A and VINKEMEIER U, 2011. STAT1 Signaling is not regulated by a Phosphorylation-Acetylation Switch. Molecular and Cellular Biology. 31(14), 3029-37 DROESCHER, M., BEGITT, A., MARG, A., ZACHARIAS, M. and VINKEMEIER, U., 2011. Cytokine-induced paracrystals prolong the activity of signal transducers and activators of transcription (STAT) and provide a model for the regulation of protein solubility by small ubiquitin-like modifier (SUMO) Journal of Biological Chemistry. 286(21), 18731-18746 NARDOZZI, JONATHAN, WENTA, NIKOLA, YASUHARA, NORIKO, VINKEMEIER, UWE and CINGOLANI, GINO, 2010. Molecular basis for the recognition of phosphorylated STAT1 by importin alpha5. Journal of Molecular Biology. 402(1), 83-100 MEYER T and VINKEMEIER U, 2010. Assessing Sequence-Specific Dna Binding And Transcriptional Activity Of Stat1 Transcription Factor. Methods In Molecular Biology (Clifton, N.J.). 647, 139-59 WENTA, N., STRAUSS, H., MEYER, S. and VINKEMEIER, U., 2008. Tyrosine phosphorylation regulates the partitioning of STAT1 between different dimer conformations Proceedings of the National Academy of Sciences of the United States of America. 105(27), 9238-9243 SPULER, SIMONE, CARL, MIRIAM, ZABOJSZCZA, JOANNA, STRAUB, VOLKER, BUSHBY, KATE, MOORE, STEVEN A, B?HRING, SYLVIA, WENZEL, KATRIN, VINKEMEIER, UWE and ROCKEN, CHRISTOPH, 2008. Dysferlin-deficient muscular dystrophy features amyloidosis. Annals of Neurology. 63(3), 323-8 MARG A, MEYER T, VIGNERON M and VINKEMEIER U, 2008. Microinjected Antibodies Interfere With Protein Nucleocytoplasmic Shuttling By Distinct Molecular Mechanisms. Cytometry. Part A : The Journal Of The International Society For Analytical Cytology. 73A(12), 1128-40 MEYER, THOMAS, BEGITT, ANDREAS and VINKEMEIER, UWE, 2007. Green fluorescent protein-tagging reduces the nucleocytoplasmic shuttling specifically of unphosphorylated STAT1. The FEBS Journal. 274(3), 815-26 MEYER, THOMAS and VINKEMEIER, UWE, 2007. STAT nuclear translocation: potential for pharmacological intervention. Expert Opinion on Therapeutic Targets. 11(10), 1355-65 L?DIGE, I., MARG, A., WIESNER, B., MALECOVá, B., OELGESCHL?GER, T. and VINKEMEIER, U, 2005. Nuclear export determines the cytokine sensitivity of STAT transcription factors. Journal of Biological Chemistry. 280(52), 43087-43099 VINKEMEIER, UWE and MEYER, THOMAS, 2005. Antiviral activity of oligomerization-deficient Stat1. Genome informatics. International Conference on Genome Informatics. 16(1), 44-8 MARG, A., SHAN, Y., MEYER, T., MEISSNER, T., BRANDENBURG, M. and VINKEMEIER, U, 2004. Nucleocytoplasmic shuttling by nucleoporins Nup153 and Nup214 and CRM1-dependent nuclear export control the subcellular distribution of latent Stat1. Journal of Cell Biology. 165(6), 823-833 MEISSNER, TORSTEN, KRAUSE, EBERHARD, L?DIGE, INGA and VINKEMEIER, UWE, 2004. Arginine methylation of STAT1: a reassessment. Cell. 119(5), 587-9; discussion 589-590 MEYER, THOMAS, HENDRY, LISA, BEGITT, ANDREAS, JOHN, SUSAN and VINKEMEIER, UWE, 2004. A single residue modulates tyrosine dephosphorylation, oligomerization, and nuclear accumulation of stat transcription factors. Journal of Biological Chemistry. 279(18), 18998-9007 VINKEMEIER, UWE, 2004. Getting the message across, STAT! Design principles of a molecular signaling circuit. Journal of Cell Biology. 167(2), 197-201