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SAXTON J, FERJENTSIK Z, DUCKER C, JOHNSON AD and SHAW PE, 2016. Stepwise evolution of Elk-1 in early deuterostomes. The FEBS journal. 283(6), 1025-38 ROSATI R, PATKI M, CHARI V, DAKSHNAMURTHY S, MCFALL T, SAXTON J, KIDDER BL, SHAW PE and RATNAM M, 2016. The Amino-terminal Domain of the Androgen Receptor Co-opts Extracellular Signal-regulated Kinase (ERK) Docking Sites in ELK1 Protein to Induce Sustained Gene Activation That Supports Prostate Cancer Cell Growth. The Journal of biological chemistry. 291(50), 25983-25998 GALBRAITH, M.D., SAXTON, J., LI, L., SHELTON, S.J., ZHANG, H., ESPINOSA, J.M. and SHAW, P.E., 2013. ERK phosphorylation of MED14 in promoter complexes during mitogen-induced gene activation by Elk-1 Nucleic Acids Research. 41(22), 10241-53 FOXLER, D.E., JAMES, V., SHELTON, S.J., VALLIM, T.Q.D.A., SHAW, P.E. and SHARP, T.V., 2011. PU.1 is a major transcriptional activator of the tumour suppressor gene LIMD1 FEBS Letters. 585(7), 1089-1096 ABRAR HUSSAIN, PETER E SHAW and JONATHAN D HIRST, 2011. Molecular dynamics simulations and in silico peptide ligand screening of the Elk-1 ETS domain. Journal of Cheminformatics. 3(1), 49 EVANS, E.L., SAXTON, J., SHELTON, S.J., BEGITT, A., HOLLIDAY, N.D., HIPSKIND, R.A. and SHAW, P.E., 2011. Dimer formation and conformational flexibility ensure cytoplasmic stability and nuclear accumulation of Elk-1 Nucleic Acids Research. 39(15), 6390-6402 LI, L., CHEUNG, S-H., EVANS, E.L. and SHAW, P.E., 2010. Modulation of gene expression and tumour cell growth by redox modification of STAT3 Cancer Research. 70(20), 8222-8232 SHAW, P.E., 2010. Could STAT3 provide a link between respiration and cell cycle progression? Cell Cycle. 9, 4294-6 LI, L, SAXTON, J and SHAW, P.E., 2010. Post-translational control of ETS transcription factors: detection of modified factors at target gene promoters.. In: PAUL J. HIGGINS, ed., Methods in Molecular Biology: Transcription Factors 647. Springer Protocols. 279-89 ZHANG, H.M, VOUGIER, S, HODGSON, G and SHAW, P.E., 2010. Integration of protein kinases into transcription complexes: identifying components of immobilised in vitro pre-initiation complexes. In: PAUL J. HIGGINS, ed., Methods in Molecular Biology: Transcription Factors 647. Springer Protocols. 291-303 ZHANG, H.M., LI, L., PAPADOPOULOU, N., HODGSON, G., EVANS, E., GALBRAITH, M., DEAR, M., VOUGIER, S., SAXTON, J. and SHAW, P.E., 2008. Mitogen-induced recruitment of ERK and MSK to SRE promoter complexes by ternary complex factor Elk-1 Nucleic Acids Research. 36(8), 2594-2607 VINCENT, E, SAXTON, J, BAKER-GLENN, C, MOAL, I, HIRST, J D, PATTENDEN, G and SHAW, P E, 2007. Effects of ulapualide A and synthetic macrolide analogues on actin dynamics and gene regulation. Cellular and Molecular Life Sciences. 64(4), 487-97 SHAW, PETER E, 2007. Peptidyl-prolyl cis/trans isomerases and transcription: is there a twist in the tail? EMBO Reports. 8(1), 40-5 MEVILLE JL, MOAL IH, BAKER-GLENN C, SHAW PE, PATTENDEN G and HIRST JD, 2007. The structural determinants of macrolide-actin binding: in silico insights Biophys. J.. 92(11), 3862-7 LI, L. and SHAW, P.E., 2006. Elevated activity of STAT3C due to higher DNA binding affinity of phosphotyrosine dimer rather than covalent dimer formation Journal of Biological Chemistry. 281(44), 33172-33181 LI, L. and SHAW, P.E., 2004. A STAT3 dimer formed by inter-chain disulphide bridging during oxidative stress Biochemical and Biophysical Research Communications. 322(3), 1005-1011