许恒 - 四川大学 - 华西基础医学与法医学院

个人简介

许恒，2004年毕业于南京大学生命科学院获得学士学位，2010年在中科学上海生命科学院获得生物技术与医药博士学位。2010年-2014年在St. Jude儿童研究医院从事博士后研究工作，2014年至今受聘于四川大学，任研究员，博士生导师。

研究领域

对相关的基础生物学实验、生物信息学分析、生物统计学分析，以及基础医学（如药理学)进行交叉学科转化研究。

近期论文

查看导师新发文章（温馨提示：请注意重名现象，建议点开原文通过作者单位确认）

1.S Yang, W Zhang,…H Xu(#), J Hu(#), Prognostic Significance of Frequent CLDN18-ARHGAP26/6 Fusion in Gastric Signet-Ring Cell Cancer.Nat Commun. 2018, 9:2447. 2.H Xu, GW Robinson, J Huang, JY Lim, et al, Common Variants in ACYP2 Influence Susceptibility to Cisplatin-induced Hearing Loss.Nat Genet.2015. 47(3): p. 263-6. 3.H Xu, C Cheng, M Devidas, D Pei, et al, ARID5B genetic polymorphisms contribute to racial disparities in the incidence and treatment outcome of childhood acute lymphoblastic leukemia.J Clin Oncol.2012. 30(7): p. 751-7. 4.H Xu, W Yang, V Perez-Andreu, M Devidas, et al, Novel susceptibility variants at 10p12.31-12.2 for childhood acute lymphoblastic leukemia in ethnically diverse populations.J Natl Cancer Inst.2013. 105(10): p. 733-42. 5.H Xu, P Wang, Y Fu, Y Zheng, et al, Length of the ORF, position of the first AUG and the Kozak motif are important factors in potential dual-coding transcripts.Cell Res.2010. 20(4): p. 445-57. 6.H Xu, H Zhang, Wenjian Yang, et al, Inherited Coding Variants at the CDKN2A Locus Influence Susceptibility to Acute Lymphoblastic Leukemia in Children.Nat Commun.2015; 6: 7553. 7. V Perez-Andreu(#), KG Roberts(#),H Xu(#), C Smith, et al, A genome-wide association study of susceptibility to acute lymphoblastic leukemia in adolescents and young adults.Blood.2015. 125(4): p. 680-6. (co-first author) 8. V Perez-Andreu, KG Roberts, RC Harvey, W Yang, C Cheng, D Pei,H Xu, et al, Inherited GATA3 variants are associated with Ph-like childhood acute lymphoblastic leukemia and risk of relapse.Nat Genet.2013. 45(12): p. 1494-8. 9. V Perez-Andreu,H Xu, and JJ Yang, The novel susceptibility variants for childhood acute lymphoblastic leukemia.J Natl Cancer Inst.2013. 105(19): p. 1512-3. 10. Z Zhang, L Zhou, L Hu, Y Zhu,H Xu, et al, Nonsense-mediated decay targets have multiple sequence-related features that can inhibit translation.Mol Syst Biol.2010. 6: p. 442.