Chaytor, Andrew - Durham University - School of Medicine, Pharmacy & Health, Medicine

个人简介

He gained his PhD in Cardiovascular Physiology and Pharmacology at the University of Wales College of Medicine, Cardiff investigating mechanisms of cellular communication in arteries and the chaos and nonlinear dynamics of blood vessel tone. Previously Andrew worked as a British Heart Foundation Research Fellow and Lecturer at the Wales Heart Research Institute, Wales College of Medicine, Cardiff and as a Senior Lecturer in Physiology at the School of Pharmacy, University of Sunderland.

研究领域

Admissions and Recruitment to Medical Schools Simulation in UG Medicine Use of a Conscientiousness Index as a measure of Professionalism Substance misuse in the undergraduate medical curriculum Mechanisms of arterial vasodilation Role of gap junctions in the cardiovascular system

近期论文

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Chaytor, A. (2015). Control of cardiac function: an overview. Anaesthesia and Intensive Care Medicine 16(5): 209-211. Nazar, H., Nazar, M., Rothwell, C., Portlock, J., Chaytor, A. & Husband, A. (2015). Teaching safe prescribing to medical students: Perspectives in the UK. Advances in Medical Education and Practice 6: 279-295. Swamy, M., Sawdon, M., Chaytor, A., Barbaro-Brown, J. & McLachlan, J. (2014). A study to investigate the effectiveness of SimMan? as an adjunct in teaching preclinical skills to medical students. BMC Medical Education 14: 231. Notley, C., Goodair, C., Chaytor, A., Carroll, J., Ghodse, H. & Kopelman, P. (2014). Report of the substance misuse in the undergraduate medical curriculum project in England. Drugs: Education, Prevention & Policy 21(2): 173-176. Carroll, J., Goodair, C., Chaytor, A., Notley, C., Ghodse, H. & Kopelman, P. (2014). Substance misuse teaching in undergraduate medical education. BMC Medical Education 14(1): 34. Chaytor, A.T., Spence, J., Armstrong, A. & McLachlan, J.C. (2012). Do students learn to be more conscientious at medical school?. BMC Medical Education 12: 54. Chaytor, AT (2009). Control of Cardiac Function: An Overview. Anaesthesia & Intensive Care Medicine 10(8): 370-72. Chaytor, AT, Gray, C, Arrowsmith, C & Tan, I (2008). Effects of ascorbic acid on phenylephrine-evoked constriction and relaxation responses to acetylcholine in aorta from aged rats. Basic and Clinical Pharmacology and Toxicology 102: 26. Edwards DH, Chaytor AT, Bakker LM & Griffith TM. (2007). Modulation of gap-junction-dependent arterial relaxation by ascorbic acid. Journal of Vascular Research 44(5): 410-422. Griffith TM, Chaytor AT, Bakker LM & Edwards DH. (2005). 5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation. Proc Natl Acad Sci U S A 102(19): 7008-7013. Chaytor AT, Bakker LM, Edwards DH & Griffith TM. (2005). Connexin-mimetic peptides dissociate electrotonic EDHF-type signalling via myoendothelial and smooth muscle gap junctions in the iliac artery. British Journal of Pharmacology 144(1): 108-114. Gavazza F, Daverio F, Chaytor AT, Griffith TM & McGuigan C. (2005). Phosphoramidate derivatives of 2',5'-dideoxyadenosine as potential inhibitors of the EDHF phenomenon. Nucleosides Nucleotides Nucleic Acids 24(5-7): 553-555. Griffith TM, Chaytor AT, Edwards DH, Daverio F & McGuigan C. (2004). Enhanced inhibition of the EDHF phenomenon by a phenyl methoxyalaninyl phosphoramidate derivative of dideoxyadenosine. British Journal of Pharmacology 142(1): 27-30. Griffith TM, Chaytor AT & Edwards DH. (2004). The obligatory link: role of gap junctional communication in endothelium-dependent smooth muscle hyperpolarization. Pharmacol Res 49(6): 551-564. Ujiie H, Chaytor AT, Bakker LM & Griffith TM. (2003). Essential role of Gap junctions in NO- and prostanoid-independent relaxations evoked by acetylcholine in intracerebral arteries. Stroke 34(2): 544-550. Griffith TM, Chaytor AT, Taylor HJ, Giddings BD & Edwards DH. (2002). cAMP facilitates EDHF-type relaxations in conduit arteries by enhancing electrotonic conduction via gap junctions. Proc Natl Acad Sci U S A 99(9): 6392-7. Chaytor AT, Taylor HJ & Griffith TM. (2002). Gap junction-dependent and -independent EDHF-type relaxations may involve smooth muscle cAMP accumulation. AJP - Heart and Circulatory Physiology 282(4): H1548-5. Taylor HJ, Chaytor AT, Edwards DH & Griffith TM. (2001). Gap junction-dependent increases in smooth muscle cAMP underpin the EDHF phenomenon in arteries. Biochemical and Biophysical Research Communications 283(3): 583-9. Chaytor AT, Martin PE, Edwards DH & Griffith TM. (2001). Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the hepatic artery. AJP - Heart and Circulatory Physiology 280(6): H2441-50. Chaytor AT, Marsh WL, Hutcheson IR & Griffith TM. (2000). Comparison of glycyrrhetinic acid isoforms and carbenoxolone as inhibitors of EDHF-type relaxations mediated via gap junctions. Endothelium 7(4): 265-78. Hutcheson IR, Chaytor AT, Evans WH & Griffith TM (1999). Nitric oxide-independent relaxations to acetylcholine and A23187 involve different routes of heterocellular communication. Role of Gap junctions and phospholipase A2. Circulation Research 84(1): 53-63. Dora KA, Martin PE, Chaytor AT, Evans WH, Garland CJ & Griffith TM. (1999). Role of heterocellular Gap junctional communication in endothelium-dependent smooth muscle hyperpolarization: inhibition by a connexin-mimetic peptide. Biochemical and Biophysical Research Communications 254(1): 27-31. Chaytor AT, Martin PE, Evans WH, Randall MD & Griffith TM. (1999). The endothelial component of cannabinoid-induced relaxation in mesenteric artery depends on gap junctional communication. Journal of Physiology 520(2): 539-50. Chaytor AT, Evans WH & Griffith TM. (1998). Central role of heterocellular gap junctional communication in endothelium-dependent relaxations of rabbit arteries. Journal of Physiology 508(2): 561-73. Chaytor AT, Evans WH & Griffith TM. (1997). Peptides homologous to extracellular loop motifs of connexin 43 reversibly abolish rhythmic contractile activity in arteries. Journal of Physiology 503(1): 99-110.