Thoresen, Marianne - University of Bristol - School of Clinical Sciences

研究领域

Physiology and neuroprotection of the newborn

近期论文

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Elstad, M, Liu, X & Thoresen, M, 2016, ‘Heart rate response to therapeutic hypothermia in infants with hypoxic-ischaemic encephalopathy’. Resuscitation, vol 106., pp. 53-57 Sabir, H, Osredkar, D, Maes, EPJ, Wood, TR & Thoresen, M, 2016, ‘Xenon combined with therapeutic hypothermia is not neuroprotective after severe hypoxia-ischemia in neonatal rats’. PLoS ONE, vol 11. Kochanek, P, Kitagawa, RS, Batchelor, P & Thoresen, M, 2016, ‘Central Nervous System Injury and Temperature Management’. Wood, T, Smit, E, Maes, E, Osredkar, D, Falck, M, Elstad, M & Thoresen, M, 2016, ‘Monitoring of cerebral blood flow during hypoxia-ischemia and resuscitation in the neonatal rat using laser speckle imaging’. Physiological reports, vol 4. Wood, T, Osredkar, D, Puchades, M, Maes, E, Falck, M, Flatebø, T, Walløe, L, Sabir, H & Thoresen, M, 2016, ‘Treatment temperature and insult severity influence the neuroprotective effects of therapeutic hypothermia’. Scientific Reports, vol 6. Sabir, H, Wood, TR, Gill, H, Liu, X, Dingley, J & Thoresen, M, 2016, ‘Xenon depresses aEEG background voltage activity whilst maintaining cardiovascular stability in sedated healthy newborn pigs’. Journal of the Neurological Sciences, vol 363., pp. 140-144 Smit, E, Liu, X, Gill, H, Jary, S, Wood, T & Thoresen, M, 2015, ‘The effect of resuscitation in 100% oxygen on brain injury in a newborn rat model of severe hypoxic-ischaemic encephalopathy’. Resuscitation, vol 96., pp. 214-9 Thoresen, M, Chakkarapani, E, Walloe, L, Loberg, E, Maes, E, Liu, X & Osredkar, D, 2015, ‘No cardiac protection from hypothermia or xenon inhalation after hypoxia-ischemia in newborn pigs’. Wood, T, Osredkar, D, Flatebo, T, Falck, M, Maes, E & Thoresen, M, 2015, ‘Temperature Dependence Of Hypothermic Neuroprotection in a Rat Model of Neonatal Hypoxic Ischaemic Enchephopathy’. Falck, M, Osredkar, D & Thoresen, M, 2015, ‘Hypothermia does not reverse cellular responses caused by lipopolysaccharide in neonatal hypoxic ischemic brain injury’.