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Born in Australia, Dr Thomas graduated from the Royal Melbourne Institute of Technology University. Her early interests were in Pharmacology, Toxicology, Organ Transplantation and, eventually, Vascular Biology (performing basic, pre-clinical and clinical research, and various projects for Industry). In 2003 she obtained a PhD in Biomedical Sciences (“Targeted delivery of anti-restenotic agents”) with Professors Julie and Gordon Campbell at The University of Queensland and Industry partner Agen Biomedical. She worked for a short time at Agen before taking up a CJ Martin Postdoctoral Fellowship from the Australian National Health and Research Council (NHMRC), held in the United Kingdom at the Bristol Heart Institute, University of Bristol and in Australia at the Australian Institute for Bioengineering and Nanotechnology, investigating the use of gene therapy in vein graft disease and the use of nanoparticles to aid targeted-drug delivery. She has returned to the University of Bristol to continue her research investigating the role of inflammation and thrombosis in Cardiovascular Disease

研究领域

Cardiovascular Disease

近期论文

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Thomas, A, Angelini, G & Zakkar, M, 2016, ‘Strategies to extend the life of saphenous vein grafts’. in: IC Tintoiu, MJ Underwood, SP Cook, H Kitabata, A Abbas (eds) Coronary Graft Failure: State of the Art. Springer, Switzerland, pp. 585-597 Tsaousi, A, Hayes, E, Di Gregoli, K, Bond, A, Bevan, L, Thomas, A & Newby, A, 2016, ‘Plaque size is decreased but M1 macrophage polarization and rupture related metalloproteinase expression are maintained after deleting T-bet in ApoE null mice’. PLoS ONE, vol 11., pp. e0148873 Thomas, A, 2016, ‘Targeting therapies to treat restenosis and vein graft disease’. in: IC Tintoiu, MJ Underwood, SP Cook, H Kitabata, A Abbas (eds) Coronary Graft Failure – State of the Art. Springer, Switzerland, pp. 715-723 Thomas, A, Eijgelaar, W, Daemen, M & Newby, A, 2015, ‘Genomic profile of foamy and non-foamy granuloma macrophages from fat-fed ApoE null mice or control mice fed a normal diet’. NCBI Thomas, AC, Eigelaar, W, Daemen, M & Newby, AC, 2015, ‘The pro-fibrotic and anti-inflammatory foam cell macrophage paradox’. Genomics Data, vol 6., pp. 136-138 Hayes, EM, Tsaousi, A, Di Gregoli, K, Jenkinson, SR, Bond, AR, Johnson, JL, Bevan, LA, Thomas, AC & Newby, AC, 2015, ‘Classical and alternative activation and metalloproteinase expression occurs in foam cell macrophages in male and female ApoE null mice in the absence of T and B lymphocytes’. in: Charles Mills, Laurel Lenz, Klaus Ley (eds) Frontiers in Immunology: M1/M2 Macrophages: The Arginine Fork in the Road to Health and Disease., pp. 147 Newby, A & Thomas, A, 2015, ‘Foam cell formation in vivo is pro-fibrotic’. in: Atherosclerosis., pp. e81-e82 Mills, CD, Thomas, AC, Lenz, LL & Munder, M, 2015, ‘Macrophage: SHIP of immunity’. in: Charles Mills, Laurel Lenz, Klaus Ley (eds) M1/M2 Macrophages: The Arginine Fork in the Road to Health and Disease. Frontiers Media, USA, pp. 42-46 Thomas, AC & Mattila, J, 2015, ‘’Of mice and men’: Arginine metabolism in human macrophages’. in: Charles Mills, Laurel Lenz, Klaus Ley (eds) M1/M2 macrophages: The arginine fork in the road to health and disease. Frontiers Media, USA, pp. 73-79 Mattila, J & Thomas, AC, 2015, ‘Nitric oxide synthase: Non-canonical expression patterns’. in: Charles Mills, Laurel Lenz, Klaus Ley (eds) M1/M2 macrophages: The arginine fork in the road to health and disease. Frontiers Media, USA, pp. 170-174

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