近期论文

查看导师新发文章 （温馨提示：请注意重名现象，建议点开原文通过作者单位确认）

Lyon, CA, Wadey, KS & George, SJ, 2015, ‘Soluble N-cadherin: A novel inhibitor of VSMC proliferation and intimal thickening’. Current Vascular Pharmacology. Lyon, CA, Johnson, JL, White, S, Sala-Newby, GB & George, SJ, 2014, ‘EC4, a truncation of soluble N-cadherin, reduces vascular smooth muscle cell apoptosis and markers of atherosclerotic plaque instability’. Molecular Therapy - Methods & Clinical Development , vol 1. Monk, BA, Williams, H, Mill, CEJ, Lyon, CA, Johnson, JL, Angelini, GD & George, SJ, 2014, ‘DYSFUNCTIONAL WNT SIGNALLING IN AGEING: IMPLICATIONS FOR CARDIOVASCULAR DISEASE’., pp. E6-E6 Lyon, CA, Johnson, JL, White, SJ, Sala-Newby, G & George, SJ, 2014, ‘EC4, a Truncation of Soluble N-cadherin Reduces Vascular Smooth Muscle Cell Apoptosis and Markers of Atherosclerotic Plaque Instability.’. Molecular Therapy - Methods & Clinical Development , vol 1. Lyon, CA, Johnson, JL, White, SJ, Sala-Newby, GM & George, SJ, 2012, ‘EC4-Fc, a similar to 50 kDa fragment of N-cadherin, reduces apoptosis and promotes atherosclerotic plaque stability’., pp. 313-314 Tsaousi, A, Williams, H, Lyon, CA, Taylor, V, Swain, A, Johnson, JL & George, SJ, 2011, ‘Wnt4/β-catenin signaling induces VSMC proliferation and is associated with intimal thickening’. Circulation Research, vol 108., pp. 427 - 436 Lyon, C, Mill, C, Tsaousi, A, Williams, H & George, S, 2011, ‘Regulation of VSMC behavior by the cadherin-catenin complex’. Frontiers in bioscience-Landmark, vol 16., pp. 644-657 Lyon, C, Koutsouki, E, Aguilera, C, Blaschuk, O & George, S, 2010, ‘Inhibition of N-cadherin retards smooth muscle cell migration and intimal thickening via induction of apoptosis’. J Vasc Surg, vol 52(5)., pp. 1301 - 1309 Tsaousi, A, Lyon, C, Williams, H & George, SJ, 2010, ‘Role of Wnt pathway in regulation of VSMC proliferation and intimal thickening’., pp. S90-S90 Lyon, C, Johnson, J, Williams, H, Sala-Newby, G & George, S, 2008, ‘Soluble N-Cadherin Overexpression Reduces Features of Atherosclerotic Plaque Instability’. Arteriosclerosis, Thrombosis, and Vascular Biology, vol 29., pp. 195 - 201