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Cheng, H, James, AF, Boyett, MR & Hancox, JC, 2016, ‘Cardiac background sodium current: Elusive but important’. Channels. Cheng, H, Li, J, James, AF, Inada, S, Choisy, SCM, Orchard, CH, Zhang, H, Boyett, MR & Hancox, JC, 2016, ‘Characterization and influence of cardiac background sodium current in the atrioventricular node’. Journal of Molecular and Cellular Cardiology, vol 97., pp. 114-124 Gadeberg, HC, Bond, RC, Kong, C, Chanoit, G, Ascione, R, Cannell, M & James, A, 2016, ‘Heterogeneity of T-Tubules in Pig Hearts’. PLoS ONE, vol 11. Hancox, JC, James, AF, Marrion, NV, Zhang, H & Thomas, D, 2016, ‘Novel ion channel targets in atrial fibrillation’. Expert Opinion on Therapeutic Targets., pp. 1-12 Walsh, MA, Stuart, AG, Schlecht, H, James, AF, Hancox, JC & Newbury-Ecob, RA, 2016, ‘Compound Heterozygous Triadin Mutation Causing Cardiac Arrest in Two Siblings’. PACE. Zhang, H, Cannell, MB, Kim, SJ, Watson, JJ, Norman, R, Calaghan, SC, Orchard, C & James, AF, 2015, ‘Cellular Hypertrophy and Increased Susceptibility to Spontaneous Calcium-Release of Rat Left Atrial Myocytes Due to Elevated Afterload’. PLoS ONE, vol 10. Choisy, SC, Cheng, H, Orchard, CH, James, AF & Hancox, JC, 2015, ‘Electrophysiological properties of myocytes isolated from the mouse atrioventricular node: L-type ICa, IKr, If, and Na-Ca exchange’. Physiological reports, vol 3. Gadeberg, HC, Bryant, SM, James, AF & Orchard, CH, 2015, ‘Altered Na/Ca exchange distribution and activity in ventricular myocytes from failing hearts’. AJP- Heart and Circulatory Phisiology, vol 310., pp. H262-H268 Bryant, SM, Kong, CHT, Watson, J, Cannell, MB, James, AF & Orchard, CH, 2015, ‘Altered distribution of ICa impairs Ca release at the t-tubules of ventricular myocytes from failing hearts’. Journal of Molecular and Cellular Cardiology, vol 86., pp. 23?31 Choisy, SC, Kim, S-J, Hancox, JC, Jones, SA & James, AF, 2015, ‘Effects of candesartan, an angiotensin II receptor type I blocker, on atrial remodeling in spontaneously hypertensive rats’. Physiological reports, vol 3.

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