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个人简介

Dr Karsten Schrobback is Postdoctoral Research Fellow and Deputy Group leader of the Cartilage Regeneration Laboratory (CRL) at the Institute of Health and Biomedical Innovation. Dr Schrobback’s areas of expertise are best described as the study of the effect of biomechanical stimulation on cartilage cells (chondrocytes), as well as cell-matrix interactions in three-dimensional cell culture systems, with the overall goal to identify critical factors of cell and tissue homeostasis in healthy and osteoarthritic cartilage.

研究领域

His main areas of research are: Biomechanical stimulation of cells Chondrocyte biology 3D cell culture models

近期论文

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Schrobback, Karsten, Klein, Travis Jacob, & Woodfield, Tim B. F. (2015) The importance of connexin hemichannels during chondroprogenitor cell differentiation in hydrogel versus microtissue culture models. Tissue Engineering Part A, 21(11-12), pp. 1785-1794. Levett, Peter A., Melchels, Ferry, Schrobback, Karsten, Hutmacher, Dietmar, Malda, Jos, & Klein, Travis J. (2014) A biomimetic extracellular matrix for cartilage tissue engineering centered on photocurable gelatin, hyaluronic acid and chondroitin sulfate. Acta Biomaterialia, 10(1), pp. 214-223. Levett, Peter, Melchels, Ferry, Schrobback, Karsten, Hutmacher, Dietmar, Malda, Jos, & Klein, Travis (2014) Chondrocyte redifferentiation and construct mechanical property development in single-component photocrosslinkable hydrogels. Journal of Biomedical Materials Research - Part A, 102(8), pp. 2544-2553. Schrobback, Karsten, Wrobel, Jana, Hutmacher, Dietmar, Woodfield, Tim B. F., & Klein, Travis J. (2013) Stage-specific embryonic antigen-4 is not a marker for chondrogenic and osteogenic potential in cultured chondrocytes and mesenchymal progenitor cells. Tissue Engineering Part A, 19(11&12), pp. 1316-1326. Jeon, June, Schrobback, Karsten, Meinert, Christoph, Sremek, Viviana, Hutmacher, Dietmar, & Klein, Travis (2013) Effect of preculture and loading on expression of matrix molecules, matrix metalloproteinases, and cytokines by expanded osteoarthritic chondrocytes. Arthritis and Rheumatism, 65(9), pp. 2356-2367.

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