个人简介
Dr. John Power is a Senior Lecturer within the Department of Physiology and the Neuroscience Honours Coordinator. He leads Neuroplasticity in Memory and Addiction Group within the Translational Neuroscience Facility. Dr. Power is an expert in the cellular basis of memory formation. His previous research has contributed to our understanding of the biophysical changes that underlie aging and associative learning, the role of the endoplasmic reticulum in neuronal calcium signalling, and amygdala neurophysiology. His group continues to investigate the cellular mechanisms of memory formation using a combination of electrophysiological recordings, live-cell fluorescent calcium imaging and genetic manipulations. The current focus of his research is the identification of novel cellular mechanisms that modulate or preserve neuronal connections that may be translated into treatments for disorders of impaired or aberrant connectivity such as Alzheimer’s disease, stroke, post-traumatic stress disorder, epilepsy and addiction.
近期论文
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Stefen H; Chaichim C; Power J; Fath T, 2016, 'Regulation of the Postsynaptic Compartment of Excitatory Synapses by the Actin Cytoskeleton in Health and Its Disruption in Disease', NEURAL PLASTICITY, vol. 2016, http://dx.doi.org/10.1155/2016/2371970
Keshavarzi S; Power JM; Albers EHH; Sullivan RKS; Sah P, 2015, 'Dendritic Organization of Olfactory Inputs to Medial Amygdala Neurons', The Journal of Neuroscience, vol. 35, no. 38, pp. 13020 - 13028, http://dx.doi.org/10.1523/JNEUROSCI.0627-15.2015
Harasta AE; Power JM; von Jonquieres G; Karl T; Drucker DJ; Housley GD; Schneider M; Klugmann M, 2015, 'Septal Glucagon-Like Peptide 1 Receptor Expression Determines Suppression of Cocaine-Induced Behavior', Neuropsychopharmacology, http://dx.doi.org/10.1038/npp.2015.47
Power JM; Sah P, 2014, 'Dendritic spine heterogeneity and calcium dynamics in basolateral amygdala principal neurons', Journal of Neurophysiology, vol. 112, no. 7, pp. 1616 - 1627, http://dx.doi.org/10.1152/jn.00770.2013
Cheng DT; Meintjes EM; Stanton ME; Desmond JE; Pienaar M; Dodge NC; Power JM; Molteno CD; Disterhoft JF; Jacobson JL; Jacobson SW, 2014, 'Functional MRI of cerebellar activity during eyeblink classical conditioning in children and adults', Human Brain Mapping, vol. 35, no. 4, pp. 1390 - 1403, http://dx.doi.org/10.1002/hbm.22261
Delaney A; Sedlak PL; Autuori E; Power J; Sah P, 2013, 'Synaptic NMDA receptors in basolateral amygdala principal neurons are triheteromeric proteins: physiological role of GluN2B subunits', Journal of Neurophysiology, vol. 109, no. 5, pp. 1391 - 1402, http://dx.doi.org/10.1152/jn.00176.2012
Kim Y; Wong ACY; Power JM; Tadros SF; Klugmann M; Moorhouse AJ; Bertrand PP; Housley GD, 2012, 'Alternative splicing of the TRPC3 ion channel calmodulin/ IP 3 receptor-binding domain in the hindbrain Enhances cation flux', The Journal of Neuroscience, vol. 32, no. 33, pp. 11414 - 11423, http://dx.doi.org/10.1523/JNEUROSCI.6446-11.2012
Delaney A; Power J; Pankaj S, 2012, 'Ifenprodil reduces excitatory synaptic transmission by blocking presynaptic P/Q type calcium channels', Journal of Neurophysiology, vol. 107, no. 6, pp. 1571 - 1575
Power J; Bocklisch C; Curby P; Pankaj S, 2011, 'Location and function of the slow afterhyperpolarization channels in the basolateral amygdala', The Journal of Neuroscience, vol. 31, no. 2, pp. 526 - 537, http://dx.doi.org/10.1523/JNEUROSCI.1045-10.2011
Oh MM; McKay BM; Power J; Disterhoft JF, 2009, 'Learning-related postburst afterhyperpolarization reduction in CA1 pyramidal neurons is mediated by protein kinase A', Proceedings of the National Academy of Sciences, vol. 106, no. 5, pp. 1620 - 1625, http://dx.doi.org/10.1073/pnas.0807708106
Faber EL; Delaney A; Power J; Sedlak PL; Crane JW; Pankaj S, 2008, 'Modulation of SK channel trafficking by beta adrenoceptors enhances excitatory synaptic transmission and plasticity in the amygdala', The Journal of Neuroscience, vol. 29, no. 43, pp. 10803 - 10813, http://dx.doi.org/10.1523/JNEUROSCI.1796-08.2008
Power J; Pankaj S, 2008, 'Competition between calcium-activated K+ channels determines cholinergic action on firing properties of basolateral amygdala projection neurons', The Journal of Neuroscience, vol. 28, no. 12, pp. 3209 - 3220, http://dx.doi.org/10.1523/JNEUROSCI.4310-07.2008
Cheng D; Disterhoft JF; Power J; Ellis DA; Desmond J, 2008, 'Neural substrates underlying human delay and trace eyeblink conditioning', Proceedings of the National Academy of Sciences, vol. 105, no. 23, pp. 8108 - 8113, http://dx.doi.org/10.1073/pnas.0800374105
Morley SN; Power JM; Coulson EJ; Bartlett PF, 2007, 'Zinc-mediated neuronal death is dependent on Trk activation', Experimental Neurology, vol. 205, no. 2, pp. 360 - 366, http://dx.doi.org/10.1016/j.expneurol.2007.02.006
Power J; Pankaj S, 2007, 'Distribution of IP3-mediated calcium responses and their role in nuclear signalling in rat basolateral amygdala neurons', The Journal of Physiology, vol. 580, pp. 835 - 857, http://dx.doi.org/10.1113/jphysiol.2006.125062
Oh MM; Wu WW; Power JM; Disterhoft JF, 2006, 'Galantamine increases excitability of CA1 hippocampal pyramidal neurons', Neuroscience, vol. 137, no. 1, pp. 113 - 123, http://dx.doi.org/10.1016/j.neuroscience.2005.08.063
Baharvand H; Power JM; Ozsarac N; Matthaei KI, 2004, 'Differentiation of embryonic stem cells into functional neuronsin vitro', Neuroscience Research Communications, vol. 35, no. 2, pp. 130 - 138, http://dx.doi.org/10.1002/nrc.20027
SAH P; FABER ESL; LOPEZ DE ARMENTIA M; POWER J, 2003, 'The Amygdaloid Complex: Anatomy and Physiology', Physiological Reviews, vol. 83, no. 3, pp. 803 - 834, http://dx.doi.org/10.1152/physrev.00002.2003
Oh MM; Power JM; Thompson LT; Disterhoft JF, 2000, 'Apamin increases excitability of CA1 hippocampal pyramidal neurons', Neuroscience Research Communications, vol. 27, no. 2, pp. 135 - 142, http://dx.doi.org/10.1002/1520-6769(200009/10)27:2<135::AID-NRC7>3.0.CO;2-X
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