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SARS-CoV-2 hijacks host CD55, CD59 and factor H to impair antibody-dependent complement-mediated lysis
Emerging Microbes & Infections ( IF 8.4 ) Pub Date : 2024-10-28 , DOI: 10.1080/22221751.2024.2417868 Laura Gebetsberger, Zahra Malekshahi, Aron Teutsch, Gabor Tajti, Frédéric Fontaine, Nara Marella, André Mueller, Lena Prantl, Hannes Stockinger, Heribert Stoiber, Anna Ohradanova-Repic
Emerging Microbes & Infections ( IF 8.4 ) Pub Date : 2024-10-28 , DOI: 10.1080/22221751.2024.2417868 Laura Gebetsberger, Zahra Malekshahi, Aron Teutsch, Gabor Tajti, Frédéric Fontaine, Nara Marella, André Mueller, Lena Prantl, Hannes Stockinger, Heribert Stoiber, Anna Ohradanova-Repic
The complement system is a vital anti-microbial defence mechanism against circulating pathogens. Excessive complement activation can have deleterious outcomes for the host and is consequently tight...
中文翻译:
SARS-CoV-2 劫持宿主 CD55、CD59 和因子 H 以损害抗体依赖性补体介导的裂解
补体系统是对抗循环病原体的重要抗菌防御机制。过度的补体激活会对宿主产生有害的结果,因此是紧密的......
更新日期:2024-10-28
中文翻译:
SARS-CoV-2 劫持宿主 CD55、CD59 和因子 H 以损害抗体依赖性补体介导的裂解
补体系统是对抗循环病原体的重要抗菌防御机制。过度的补体激活会对宿主产生有害的结果,因此是紧密的......