Stress generation posits that (a) individuals at-risk for psychopathology may inadvertently experience higher rates of prospective dependent stress (i.e., stressors that are in part influenced by their thoughts and behaviors) but not independent stress (i.e., stressors occurring outside their influence), and (b) this elevated dependent stress, in some measure, is what places these individuals at-risk for future psychopathology. In recognition of 30 years of stress generation research, we conducted a systematic review and meta-analysis using frequentist and Bayesian approaches (102 articles with 104 eligible studies, N = 31,541). Generally strong support was found for psychopathology predicting dependent stress (e.g., dsOverall psychopathology = 0.36-0.52, BF₁₀ = 946.00 to 4.65 × 10¹⁸). Moderator analyses for dependent stress revealed larger effects for briefer assessments periods, shorter follow-ups, and self-report measures than for interviews. Among risk factors, depressogenic cognitive styles (ds = .26-.50, BF₁₀ = 47.50 to 1.00 × 10⁵) and general interpersonal vulnerability (ds = .26-.44, BF₁₀ = 2.72 to 2708.00) received the strongest support as stress generation mechanisms, and current evidence is modest for protective factors predicting dependent stress. Overall, larger effects were generally found for prospective prediction of dependent stress than independent stress. Evaluations of mediation in the research literature were relatively few, limiting the current review to qualitative analysis of the mediation component of stress generation. General support was found, however, for dependent stress as a mediator for psychopathology and associated risk factors in relation to subsequent psychopathology. The current review ends with recommendations for future research and integration of stress generation within minority stress frameworks. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

中文翻译：

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对 30 年压力产生研究的系统评价和贝叶斯荟萃分析：潜在负面生活事件的临床、心理和社会人口学风险和保护因素。


压力产生假设 （a） 有精神病理学风险的个体可能会无意中经历更高的预期依赖压力（即，部分受其思想和行为影响的压力源）但不是独立压力（即，发生在他们影响之外的压力源），以及 （b） 这种升高的依赖压力，在某种程度上，是使这些人在未来面临精神病理学风险的原因。鉴于 30 年的压力生成研究，我们使用频率论和贝叶斯方法进行了系统评价和荟萃分析（102 篇文章，104 项符合条件的研究，N = 31,541）。通常发现对精神病理学预测依赖性压力有很强的支持（例如，dsOverall psychopathology = 0.36-0.52，BF₁₀ = 946.00 至 4.65 × 10¹⁸）。与访谈相比，对依赖性压力的调节者分析显示，较简短的评估期、较短的随访和自我报告措施的影响更大。在风险因素中，抑郁认知类型 （ds = .26-.50， BF₁₀ = 47.50 至 1.00 × 10⁵） 和一般人际易感性 （ds = .26-.44， BF₁₀ = 2.72 至 2708.00） 作为压力产生机制获得了最强的支持，目前预测依赖压力的保护因素的证据是适度的。总体而言，通常发现对依赖应力的前瞻性预测的影响大于独立应力。研究文献中对中介的评价相对较少，将目前的综述限制在对压力产生的中介成分的定性分析。然而，普遍支持依赖性压力作为精神病理学的中介以及与后续精神病理学相关的危险因素。 目前的综述以对未来研究和将压力产生整合到少数压力框架中的建议结束。（PsycInfo 数据库记录 （c） 2024 APA，保留所有权利）。