当前位置:
X-MOL 学术
›
J. Adv. Res.
›
论文详情
Our official English website, www.x-mol.net, welcomes your
feedback! (Note: you will need to create a separate account there.)
Loss of Nup155 promotes high fructose-driven podocyte senescence by inhibiting INO80 mRNA nuclear export
Journal of Advanced Research ( IF 11.4 ) Pub Date : 2024-08-05 , DOI: 10.1016/j.jare.2024.08.007 Li Chen 1 , Tangdi Xu 1 , Zixuan Wang 1 , Chengzhi Wang 2 , Lei Fang 2 , Lingdong Kong 1
Journal of Advanced Research ( IF 11.4 ) Pub Date : 2024-08-05 , DOI: 10.1016/j.jare.2024.08.007 Li Chen 1 , Tangdi Xu 1 , Zixuan Wang 1 , Chengzhi Wang 2 , Lei Fang 2 , Lingdong Kong 1
Affiliation
Podocyte senescence causes podocyte loss and glomerulopathy. Excessive fructose intake is a risk factor for podocyte injury. However, whether high fructose promotes podocyte senescence remains unknown.
中文翻译:
Nup155 缺失通过抑制 INO80 mRNA 核输出促进高果糖驱动的足细胞衰老
足细胞衰老导致足细胞丢失和肾小球病。过量的果糖摄入是足细胞损伤的危险因素。然而,高果糖是否促进足细胞衰老仍然未知。
更新日期:2024-08-05
中文翻译:
Nup155 缺失通过抑制 INO80 mRNA 核输出促进高果糖驱动的足细胞衰老
足细胞衰老导致足细胞丢失和肾小球病。过量的果糖摄入是足细胞损伤的危险因素。然而,高果糖是否促进足细胞衰老仍然未知。