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Nitidine chloride inhibits G2/M phase by regulating the p53/14-3-3 Sigma/CDK1 axis for hepatocellular carcinoma treatment
Heliyon ( IF 3.4 ) Pub Date : 2024-01-03 , DOI: 10.1016/j.heliyon.2024.e24012 Bo Zhang 1 , Bo Zhou 1 , Guihong Huang 2, 3, 4 , Jing'an Huang 1 , Xiaoxin Lin 1 , Zonghuai Li 1 , Yuanchu Lian 1 , Qiujie Huang 5 , Yong Ye 6, 7
Heliyon ( IF 3.4 ) Pub Date : 2024-01-03 , DOI: 10.1016/j.heliyon.2024.e24012 Bo Zhang 1 , Bo Zhou 1 , Guihong Huang 2, 3, 4 , Jing'an Huang 1 , Xiaoxin Lin 1 , Zonghuai Li 1 , Yuanchu Lian 1 , Qiujie Huang 5 , Yong Ye 6, 7
Affiliation
Liver cancer had become the sixth most common cancer. Nitidine chloride (NC) has demonstrated promising anti-HCC properties; however, further elucidation of its mechanism of action is necessary.
中文翻译:
硝替丁氯化物通过调节 p53/14-3-3 Sigma/CDK1 轴抑制 G2/M 期治疗肝细胞癌
肝癌已成为第六大常见癌症。硝替丁氯化物 (NC) 已显示出有前景的抗 HCC 特性;然而,有必要进一步阐明其作用机制。
更新日期:2024-01-03
中文翻译:
硝替丁氯化物通过调节 p53/14-3-3 Sigma/CDK1 轴抑制 G2/M 期治疗肝细胞癌
肝癌已成为第六大常见癌症。硝替丁氯化物 (NC) 已显示出有前景的抗 HCC 特性;然而,有必要进一步阐明其作用机制。