N-modified sludge-derived biochar (N-SDBC) was prepared and then used for activation of peroxydisulfate (PDS) to degrade fluoroquinolone antibiotics (FQs). Seven commonly used FQs were degraded by N-SDBC/PDS effectively, with the rate constants from 0.131 min⁻¹ to 0.505 min⁻¹. Scavenging experiments combined with electron paramagnetic resonance (EPR) confirmed that ¹O₂ was the dominated reactive species, and the radicals SO₄^•− and ^•OH played minor roles. In-depth study revealed that the graphite-N and Fe⁰ on the surface of N-SDBC were the main active sites for PDS activation. The elevated N precursors and calcination temperature enhanced the yields of graphite-N and Fe⁰. Products analysis indicated enrofloxacin (ENR) degradation by ¹O₂ with the fragmentation of the piperazine ring, the disconnection of the quinolone ring, decarboxylation, and defluorination. The toxicity assessment by Escherichia coli and ECOSAR showed that the biological toxicity of ENR could be reduced after treating by N-SDBC/PDS. The application in surface water and wastewater samples demonstrates the efficiency of FQs removal by N-SDBC/PDS.

制备了 N 改性污泥衍生生物炭 (N-SDBC)，然后用于活化过二硫酸盐 (PDS) 以降解氟喹诺酮类抗生素 (FQs)。七种常用的 FQ 被 N-SDBC/PDS 有效降解，速率常数从 0.131 min ^-1到 0.505 min ^-1。清除实验结合电子顺磁共振(EPR) 证实¹ O ₂是主要的反应物种，自由基SO ₄ ^•-和^• OH 起次要作用。深入研究揭示了石墨-N和Fe ⁰N-SDBC表面是PDS活化的主要活性位点。升高的N前体和煅烧温度提高了石墨-N和Fe ⁰的产率。产物分析表明，恩诺沙星 (ENR) 被¹ O ₂降解，哌嗪环断裂、喹诺酮环断开、脱羧和脱氟。大肠杆菌和ECOSAR的毒性评估表明，经N-SDBC/PDS处理后，ENR的生物毒性可以降低。在地表水和废水样品中的应用证明了 N-SDBC/PDS 去除 FQ 的效率。