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FAM135B sustains the reservoir of Tip60-ATM assembly to promote DNA damage response
Clinical and Translational Medicine ( IF 7.919 ) Pub Date : 2022-08-17 , DOI: 10.1002/ctm2.945
Kai Zhang 1 , Qingnan Wu 1, 2 , Wenzhong Liu 1 , Yan Wang 1, 2 , Lianmei Zhao 3 , Jie Chen 1, 2 , Haoyu Liu 1 , Siqi Liu 1 , Jinting Li 1 , Weimin Zhang 1, 2, 4, 5 , Qimin Zhan 1, 2, 4, 5, 6
Clinical and Translational Medicine ( IF 7.919 ) Pub Date : 2022-08-17 , DOI: 10.1002/ctm2.945
Kai Zhang 1 , Qingnan Wu 1, 2 , Wenzhong Liu 1 , Yan Wang 1, 2 , Lianmei Zhao 3 , Jie Chen 1, 2 , Haoyu Liu 1 , Siqi Liu 1 , Jinting Li 1 , Weimin Zhang 1, 2, 4, 5 , Qimin Zhan 1, 2, 4, 5, 6
Affiliation
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Recently, the mechanism by which cells adapt to intrinsic and extrinsic stresses has received considerable attention. Tat-interactive protein 60-kDa/ataxia–telangiectasia-mutated (TIP60/ATM) axis-mediated DNA damage response (DDR) is vital for maintaining genomic integrity.
中文翻译:
FAM135B 维持 Tip60-ATM 组装的水库以促进 DNA 损伤反应
最近,细胞适应内在和外在压力的机制受到了相当多的关注。Tat 相互作用蛋白 60-kDa/共济失调-毛细血管扩张症突变 (TIP60/ATM) 轴介导的 DNA 损伤反应 (DDR) 对于维持基因组完整性至关重要。
更新日期:2022-08-22
中文翻译:

FAM135B 维持 Tip60-ATM 组装的水库以促进 DNA 损伤反应
最近,细胞适应内在和外在压力的机制受到了相当多的关注。Tat 相互作用蛋白 60-kDa/共济失调-毛细血管扩张症突变 (TIP60/ATM) 轴介导的 DNA 损伤反应 (DDR) 对于维持基因组完整性至关重要。