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Melatonergic signalling instructs transcriptional inhibition of IFNGR2 to lessen interleukin-1β-dependent inflammation
Clinical and Translational Medicine ( IF 7.919 ) Pub Date : 2022-02-20 , DOI: 10.1002/ctm2.716
Yaoyao Xia 1 , Qingzhuo Zhang 1 , Yuyi Ye 1 , Xiaoyan Wu 1 , Fang He 2 , Yuanyi Peng 2 , Yulong Yin 3 , Wenkai Ren 1
Clinical and Translational Medicine ( IF 7.919 ) Pub Date : 2022-02-20 , DOI: 10.1002/ctm2.716
Yaoyao Xia 1 , Qingzhuo Zhang 1 , Yuyi Ye 1 , Xiaoyan Wu 1 , Fang He 2 , Yuanyi Peng 2 , Yulong Yin 3 , Wenkai Ren 1
Affiliation
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Immunotransmitters (e.g., neurotransmitters and neuromodulators) could orchestrate diverse immune responses; however, the elaborated mechanism by which melatonergic activation governs inflammation remains less defined.
中文翻译：
褪黑素信号传导抑制 IFNGR2 的转录以减轻白细胞介素 1β 依赖性炎症
免疫递质（例如神经递质和神经调节剂）可以协调不同的免疫反应；然而，褪黑素能激活控制炎症的详细机制仍不清楚。
更新日期：2022-02-21
中文翻译：
褪黑素信号传导抑制 IFNGR2 的转录以减轻白细胞介素 1β 依赖性炎症
免疫递质（例如神经递质和神经调节剂）可以协调不同的免疫反应；然而，褪黑素能激活控制炎症的详细机制仍不清楚。
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