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Zhu W#, Li J#, Zhang R, Cai Y, Wang C, Qi S, Chen S, Liang X, Qi N*, Hou F* (2019) TRAF3IP3 mediates the recruitment of TRAF3 to MAVS for antiviral innate immunity. EMBO J, doi: 10.15252/embj.2019102075
Cadena C, Ahmad S, Xavier A, Willemsen J, Park S, Park J, Oh S, Fujita T, Hou F, Binder M, Hur S* (2019) Ubiquitin-Dependent and -Independent Roles of E3 Ligase RIPLET in Innate Immunity. Cell, 177, 1-14
Zhou P, Ding X, Wan X, Liu L, Yuan X, Zhang W, Hui X, Meng G, Xiao H, Li B, Zhong J, Hou F, Deng L, Zhang Y* (2018) MLL5 suppresses antiviral innate immune response by facilitating STUB1-mediated RIG-I degradation. Nat Commun, |9:1243|DOI: 10.1038/s41467-018-03563-8
Li X, Gadzinsky A, Gong L, Tong H, Calderon V, Li Y, Kitamura D, Klein U, Langdon WY, Hou F, Zou YR, Gu H* (2018) Cbl Ubiquitin Ligases Control B Cell Exit from the Germinal-Center Reaction. Immunity, 48, 530-541
Qi N#, Shi Y#, Zhang R, Zhu W, Yuan B, Li X, Wang C, Zhang X, Hou F* (2017) Multiple truncated isoforms of MAVS prevent its spontaneous aggregation in antiviral innate immune signalling. Nat Commun, |8:15676|DOI: 10.1038/ncomms15676
Shi Y#, Yuan B#, Zhu W#, Zhang R, Li L, Hao X, Chen S, Hou F* (2017) Ube2D3 and Ube2N are essential for RIG-I-mediated MAVS aggregation in antiviral innate immunity. Nat Commun, |8:15138| DOI:10.1038/ncomms 15138
Shi Y#, Yuan B#, Qi N#, Zhu W, Su J, Li X, Qi P, Zhang D, Hou F* (2015) An autoinhibitory mechanism modulates MAVS activity in antiviral innate immune response. Nat Commun, |6:78111|DOI: 10.1038/ncomms8811
Xu H, He X, Zheng H, Huang L, Hou F, Yu Z, Cruz MJ, Borkowski B, Zhang X*, Chen ZJ*, Jiang Q-X* (2014) Structural basis for the prion-like MAVS filaments in antiviral innate immunity. ELife, 3, e01489
Hou F, Sun L, Zheng H, Skaug B, Jiang QX, and Chen ZJ* (2011) MAVS Forms Functional Prion-like Aggregates to Activate and Propagate Antiviral Innate Immune Response. Cell, 146, 448-461
Chu CW, Hou F, Zhang J, Phu L, Loktev AV, Kirkpatrick DS. Jackson PK, Zhao Y, and Zou H* (2011) A novel acetylation of beta-tubulin by San modulates microtubule polymerization via down-regulating tubulin incorporation. Mol Biol Cell, 22, 448-456
Zeng W, Sun L, Jiang X, Chen X, Hou F, Adhikari A, Xu M, and Chen ZJ* (2010) Reconstitution of the RIG-I pathway reveals a signaling role of unanchored polyubiquitin chains in innate immunity. Cell, 141, 315-330
Gordillo M, Vega H, Trainer AH, Hou F, Sakai N, Luque R, Kayserili H, Basaran S, Skovby F, Hennekam RC, Uzielli ML, Schnur RE, Manouvrier S, Chang S, Blair E, Hurst JA, Forzano F, Meins M, Simola KO, Raas-Rothschild A, Schultz RA, McDaniel LD, Ozono K, Inui K, Zou H and Jabs EW* (2008) The molecular mechanism underlying Roberts syndrome involves loss of ESCO2 acetyltransferase activity. Hum Mol Genet, 17(14), 2172-80
Hou F, Chu CW, Kong X, Yokomori K, and Zou H* (2007) The acetyltransferase activity of San stabilizes the mitotic cohesin at the centromeres in a shugoshin-independent manner. J Cell Biol, 177, 587-597
Hou F, and Zou H* (2005) Two human orthologues of Eco1/Ctf7 acetyltransferases are both required for proper sister-chromatid cohesion. Mol Biol Cell, 16, 3908-3918
Jia J, Zhang L, Zhang Q, Tong C, Wang B, Hou F, Amanai K, and Jiang J* (2005) Phosphorylation by double-time/CKIepsilon and CKIalpha targets cubitus interruptus for Slimb/beta-TRCP-mediated proteolytic processing. Dev Cell, 9, 819-830
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